A sympathetic brake on gut GLP
The brain- gut neurocircuitry is proving to be finely involved in a wide range of physiological functions. In this issue of Neuron , Ren et al. show that adrenergic signaling suppresses postprandial glucagon-like peptide 1 ( GLP-1 ) secretion. This, in turn, raises circulating glucose levels and impairs brain glucose uptake and cognitive function.
The brain- gut neurocircuitry is proving to be finely involved in a wide range of physiological functions. In this issue of Neuron , Ren et al. 1 show that adrenergic signaling suppresses postprandial glucagon-like peptide 1 ( GLP-1 ) secretion. This, in turn, raises circulating glucose levels and impairs brain glucose uptake and cognitive function.
A sympathetic brake on gut GLP
The brain- gut neurocircuitry is proving to be finely involved in a wide range of physiological functions. In this issue of Neuron , Ren et al.1 show that adrenergic signaling suppresses postprandial glucagon-like peptide 1 ( GLP-1 ) secretion.
The brain- gut neurocircuitry is proving to be finely involved in a wide range of physiological functions. In this issue of Neuron , Ren et al.1 show that adrenergic signaling suppresses postprandial glucagon-like peptide 1 ( GLP-1 ) secretion. This, in turn, raises circulating glucose levels and impairs brain glucose uptake and cognitive function.
A sympathetic brake on gut GLP
The brain- gut neurocircuitry is proving to be finely involved in a wide range of physiological functions. In this issue of Neuron , Ren et al.1 show that adrenergic signaling suppresses postprandial glucagon-like peptide 1 ( GLP-1 ) secretion. This, in turn, raises circulating glucose levels and impairs brain glucose uptake and cognitive function.
A sympathetic brake on gut GLP-1 release Antonio J. López-Gambero, Victor Jouque, Daniela Cota Pages 865-867 View PDF Article preview
A sympathetic brake on gut GLP
Content The brain- gut neurocircuitry is proving to be finely involved in a wide range of physiological functions. In this issue of Neuron , Ren et al. show that adrenergic signaling suppresses postprandial glucagon-like peptide 1 ( GLP-1 ) secretion. This, in turn, raises circulating glucose levels and impairs brain glucose uptake and cognitive function.
The brain- gut neurocircuitry is proving to be finely involved in a wide range of physiological functions. In this issue of Neuron , Ren et al.1 show that adrenergic signaling suppresses postprandial glucagon-like peptide 1 ( GLP-1 ) secretion. This, in turn, raises circulating glucose levels and impairs brain glucose uptake and cognitive function.
A sympathetic brake on gut GLP
The brain- gut neurocircuitry is proving to be finely involved in a wide range of physiological functions. In this issue of Neuron , Ren et al.1 show that adrenergic signaling suppresses postprandial glucagon-like peptide 1 ( GLP-1 ) secretion.
Ren et al. characterize a neuro-enteroendocrine cell unit operating at the brain- gut neuronal axis, which restrains GLP-1 release upon glucose ingestion and compromises brain function by reducing glucose utilization in the brain.