AbCellera Announces Positive Preclinical Data for ABCL575, an Anti-OX40L Antibody for Atopic Dermatitis

Analysis reveals significant industry trends and economic implications

Release Date

2025-05-12

Category

Clinical Trial Event

Reference

Source

Breakthrough Clinical Results

AbCellera presented preclinical data on ABCL575, a half-life extended anti-OX40L monoclonal antibody for moderate-to-severe atopic dermatitis. The data showed potent inhibition of inflammatory pathways, favorable pharmacokinetics, and potential for less frequent dosing. The drug is expected to enter Phase 1 clinical trials in 2025.

Key Highlights

  • Potent inhibition of inflammatory pathways in preclinical studies.
  • Favorable pharmacokinetics and potential for less frequent dosing.
  • Positive nonclinical safety profile.
  • Expected entry into Phase 1 clinical trials in 2025.

Incidence and Prevalence

Global Epidemiology of Atopic Dermatitis

Several studies provide estimates of the incidence and prevalence of atopic dermatitis (AD) globally, with varying results depending on the methodology and year of publication. Here's a summary of some of the latest findings:

2021 Estimates:

2019 Estimates:

Other Estimates:

Key Considerations:

It's important to note that these are just a few examples, and other studies may provide different estimates. For the most up-to-date information, it's recommended to consult the latest publications and reports on AD epidemiology.

Emerging Mechanism of Action

KnolQuest, developed by Pienomial LLC, summarizes key mechanisms of action (MoA) emerging for atopic dermatitis (AD) treatment based on recent PubMed publications:

1. Targeting Type 2 Inflammation:

  • IL-4/IL-13 Inhibition: This MoA aims to block the signaling of IL-4 and IL-13, key cytokines driving type 2 inflammation in AD. Dupilumab, a monoclonal antibody targeting the shared IL-4 receptor alpha subunit, has shown significant efficacy in reducing AD signs and symptoms. Other IL-13 specific inhibitors like lebrikizumab and tralokinumab have also demonstrated promising results in clinical trials.
  • IL-31 Inhibition: IL-31 is a cytokine involved in pruritus (itch) in AD. Nemolizumab, a monoclonal antibody targeting the IL-31 receptor, has shown efficacy in reducing itch and improving skin lesions in AD.
  • OX40-OX40L Pathway Inhibition: This pathway plays a role in T-cell activation and inflammation. Rocatinlimab (anti-OX40) and amlitelimab (anti-OX40L) are being investigated for their potential to modulate T-cell responses and reduce AD severity.

2. Janus Kinase (JAK) Inhibition:

  • JAK inhibitors block the activity of JAK enzymes, which are involved in cytokine signaling and inflammation. Several oral JAK inhibitors, including baricitinib (JAK1/2), upadacitinib (JAK1), and abrocitinib (JAK1), have demonstrated efficacy in moderate-to-severe AD. Topical JAK inhibitors, such as ruxolitinib cream and delgocitinib ointment, are also being developed for milder forms of AD.

3. Phosphodiesterase 4 (PDE4) Inhibition:

  • PDE4 inhibitors block the activity of PDE4 enzymes, which are involved in the breakdown of cyclic adenosine monophosphate (cAMP), a molecule that regulates inflammation. Crisaborole, a topical PDE4 inhibitor, is approved for mild-to-moderate AD in patients 2 years and older. Difamilast, another topical PDE4 inhibitor, has shown promising results in clinical trials.

4. Aryl Hydrocarbon Receptor (AhR) Modulation:

  • Tapinarof is a topical AhR modulating agent that has shown efficacy in reducing AD signs and symptoms. The exact mechanism of action is not fully understood, but it is thought to involve modulation of immune responses and skin barrier function.

5. Restoring Skin Barrier Function:

  • EpiCeram, a ceramide-dominant barrier repair formulation, has shown efficacy in improving skin barrier function and reducing AD symptoms. It contains a combination of ceramides, cholesterol, and fatty acids that mimic the natural composition of the skin barrier.

These MoAs represent the current focus of AD research and drug development. Ongoing and future clinical trials will further elucidate their efficacy, safety, and potential role in personalized AD treatment.

Drug used in other indications

ABCL575 (Rademikibart) is primarily being investigated for atopic dermatitis (AD). While the provided text mentions numerous biologics and other treatments for AD, it doesn't specify other indications for which ABCL575 is currently being trialed, nor the intervention models for those trials. The text does mention that ABCL575 blocks IL-4Rα-mediated signal transduction, similar to dupilumab, and that it has shown promising results in a phase I trial for AD, with rapid and sustained improvements in AD severity and quality of life. However, without more information, it's impossible to determine what other conditions it might be effective for. To find out more about ongoing trials for ABCL575, it's recommended to search clinical trial databases like ClinicalTrials.gov using the drug's name or other identifiers.

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