Avidicure to Present Preclinical Data on TROP2-Targeting Antibody at ASCO 2025

Analysis reveals significant industry trends and economic implications

Release Date

2025-05-23

Category

Clinical Trial Event

Reference

Source

Breakthrough Clinical Results

Avidicure announced it will present preclinical data at the 2025 American Society of Clinical Oncology (ASCO) Annual Meeting. The data highlights AVC-S-101, a TROP2-targeting antibody, showing prolonged and significantly improved tumor control in animal models and immune system activation in non-small cell lung cancer (NSCLC) patient samples. AVC-S-101 is a first-in-class dual agonistic multifunctional antibody designed to harness the full power of the immune system. The company also announced its participation in the BIO International Convention.

Key Highlights

  • Preclinical data on AVC-S-101, a TROP2-targeting antibody, will be presented at ASCO 2025.
  • AVC-S-101 demonstrated prolonged and significantly improved tumor control in animal models.
  • AVC-S-101 showed strong activation of the immune system in human NSCLC samples.
  • Avidicure will also present at the BIO International Convention.

Emerging Mechanism of Action

Emerging Mechanisms of Action for Non-Small Cell Lung Cancer Treatment

Based on recent publications, several key mechanisms of action have emerged for the treatment of Non-Small Cell Lung Cancer (NSCLC):

Immune Checkpoint Inhibitors

PD-L1 Targeted Therapy

Anti-Angiogenic Therapy

Tyrosine Kinase Inhibitors

MET Inhibition

Recent Approvals for Early-Stage NSCLC

Recent approvals for early-stage NSCLC include: - Adjuvant osimertinib - Adjuvant atezolizumab - Adjuvant pembrolizumab - Neoadjuvant nivolumab combined with chemotherapy

These emerging mechanisms of action represent significant advances in the treatment landscape for NSCLC, offering more targeted and potentially effective therapeutic options for patients with various subtypes and stages of the disease.

Key Unmet Needs and Target Populations for Non-small Cell Lung Cancer Research

Unmet Needs

Treatment Resistance

  • Patients inevitably develop resistance to ALK inhibitors leading to tumor relapse that commonly manifests as brain metastasis
  • Several approaches aim to overcome resistance mechanisms in ALK-positive NSCLC including knowledge-based alternate use of different ALK inhibitors and combined therapies
  • Lack of prolonged disease control in most NSCLC patients despite chemotherapy and molecular-targeted therapies

Biomarker Development

  • Need for easy-to-use and robust clinical gene signatures for improved prognosis and treatment prediction
  • Low RRM1 mRNA expression in both peripheral blood and tumor tissue is associated with better response to gemcitabine/carboplatin chemotherapy
  • ERCC1 mRNA expression in tumor tissue may be a predictive and prognostic indicator in advanced NSCLC patients receiving gemcitabine/carboplatin chemotherapy
  • Need for markers to determine which patients and tumor types are most likely to respond to new treatments

Targeted Therapies

  • The role of PD-1 inhibitors in treating EGFR-mutant and ALK-rearranged lung cancer has yet to be determined
  • Limited proportion of patients benefiting from receptor tyrosine kinase (RTK) targeting agents
  • Difficulty in conducting randomized clinical trials for ALK-targeted therapies due to low frequency of ALK rearrangements (3-5% of NSCLC)

Novel Approaches

  • Epigenetic modulation could be a potential therapeutic approach to prevent metastasis formation as prometastatic phenotype was reversed due to inhibition of DNA methyltransferase
  • Traditional Chinese medicine (TCM) monomers show markedly inhibitory actions for NSCLC through M1/M2 macrophage modulation

Regional Disparities

  • Lack of comprehensive data on lung cancer in India

Target Populations

Genetic Mutation Subgroups

  • EGFR-mutant and ALK-rearranged populations
  • ALK gene rearrangements are present in approximately 3.5% of lung carcinoma and 3.7% of lung adenocarcinoma patients
  • Patients with ALK rearrangements do not respond to EGFR-specific tyrosine kinase inhibitors but benefit from ALK inhibitors
  • Patients with KRAS mutations
  • PD-L1 mutations

Rare Genetic Alterations

  • PDGFRA gene amplification was detected in a sunitinib-sensitive adenosquamous NSCLC cell line and in a small fraction of squamous cell NSCLC primary tumor specimens
  • Rare tumors showing PDGFC-mediated PDGFRA activation may be clinically responsive to pharmacologic PDGFRA or PDGFC inhibition

Disease Stage-Specific Groups

  • Patients with early-stage disease, particularly stage IA or IB adenocarcinoma
  • High-risk patients with stage I NSCLC who may benefit from adjuvant chemotherapy

Histological Subtypes

  • Squamous cell carcinoma patients of all stages

Cell-Type Specific

  • CD133-positive NSCLC cells (cancer stem cells)
  • Tumor-associated macrophages (TAM) are abundant in numerous cancer types and may serve as potential therapeutic targets

Geographic Populations

  • Indian population (where lung cancer is the fourth leading cause of cancer, 5.9% of cases, and third leading cause of cancer-related mortality)

Drug used in other indications

Based on the provided context, there is no information available about:

  1. A compound or drug called AVC-S-101
  2. Any clinical trials involving AVC-S-101
  3. Any indications for which AVC-S-101 is being trialed
  4. Any intervention models for AVC-S-101 trials

The context discusses various treatments for non-small cell lung cancer (NSCLC) and other conditions, but does not mention AVC-S-101 specifically.

Stay Ahead with More Insights

Log on to knolens for more information.