Adagene Announces Positive Updated Data for Muzastotug (ADG126) in Combination with Pembrolizumab for Colorectal Cancer at ASCO

Analysis reveals significant industry trends and economic implications

Release Date

2025-05-23

Category

Clinical Trial Event

Reference

Source

Breakthrough Clinical Results

Adagene announced updated Phase 1b/2 data for its anti-CTLA-4 antibody, Muzastotug (ADG126), in combination with pembrolizumab, in patients with microsatellite stable colorectal cancer (MSS CRC) without liver metastases. The 20 mg/kg Q6W dose showed a 29% confirmed overall response rate (ORR) with low toxicity (<20% Grade 3 adverse events). All six responders in the 20 mg/kg cohorts remain on treatment, demonstrating durable responses. The 10 mg/kg cohorts showed a median overall survival (OS) of 19.4 months. These results suggest a potential benefit of ADG126 in combination with pembrolizumab for MSS CRC.

Key Highlights

  • 29% confirmed ORR in 20 mg/kg cohorts of Muzastotug (ADG126) + pembrolizumab in MSS CRC patients without liver metastases.
  • Low toxicity profile (<20% Grade 3 adverse events) observed with the 20 mg/kg Q6W dose.
  • Durable responses observed, with all six responders in the 20 mg/kg cohorts remaining on treatment.
  • Median OS of 19.4 months in the 10 mg/kg cohorts.

Incidence and Prevalence

Global Colorectal Cancer Incidence and Prevalence

Global Incidence Patterns

Colorectal cancer is recognized as one of the most deadliest malignancies worldwide and there is an alarming uptrend in the incidence of early onset colorectal cancer (EOCRC) across the globe.

According to the International Agency for Research on Cancer (IARC) data for 1998-2002, the highest colorectal cancer incidence rates were observed in registries from: - North America - Oceania - Europe, including Eastern European countries

These high rates are likely the result of increases in risk factors associated with "Westernization," such as obesity and physical inactivity.

In contrast, the lowest colorectal cancer incidence rates were observed from registries in: - Asia - Africa - South America

In Iran, colorectal cancer is noted as one of the most common incident cancers and a common cause of cancer death.

For immigrants, particularly those born in Asia and Africa, lower rates of all site cancer incidence compared to Italian natives were observed. The lowest incidence rate ratio (IRR) for colorectal cancer was observed in: - Males from South-Central America (IRR 0.19, 95%CI 0.09-0.44) - Females from Asia (IRR 0.32, 95%CI 0.18-0.70)

Regional Variations

Asian countries have a wide range of colorectal cancer mortality-to-incidence ratio (MIR), from a minimum of 0.24 to a maximum of 0.86.

In 2012, for males, lung, prostate, and colorectal cancer in combination represented almost 30% of the cancer burden in countries that have attained very high levels of human development in the Eastern Mediterranean region.

Colon cancer is one of the cancer types showing the smallest relative variation in incidence between occupational categories.

Mortality Trends

Colorectal cancer mortality rates have declined in many longstanding as well as newly economically developed countries but continue to increase in some low-resource countries of South America and Eastern Europe.

There is limited data available on the incidence and genetic characteristics of EOCRC from resource-poor countries, particularly Africa.

Emerging Mechanism of Action

Emerging Mechanisms of Action for Colorectal Cancer

Based on recent publications, several key mechanisms of action (MoA) are emerging for colorectal cancer treatment:

Signaling Pathway Interventions

  • PF-04449913 demonstrates anti-tumor effects by blocking the ERK/p65 signaling pathway and repressing MMP9 expression, reducing colorectal cancer cell viability in a dose-dependent manner
  • B7-H4 siRNA inhibits colorectal cancer proliferation, invasion, and migration by targeting the CXCL12/CXCR4 and JAK2/STAT3 signaling pathways
  • Transforming Growth Factor-β (TGF-β) signaling plays a dual role: tumor suppressor in early stages and promoter of tumor progression in later stages
  • Wnt regulatory genes DKK1 and Wnt5a contribute to colonic carcinogenesis and are often silenced by promoter hypermethylation
  • Glucose-regulated protein 78 (GRP78) influences tumor growth through the HIF-1α/VEGF/VEGFR2 pathway

Anti-inflammatory Approaches

  • Selective cyclooxygenase-2 inhibitors (rofecoxib, celecoxib) reduce risk of colorectal neoplasia
  • Nonselective nonsteroidal anti-inflammatory drugs demonstrate protective effects against colorectal neoplasia
  • MABp1, an antibody targeting interleukin 1α, shows antitumor activity and relief of symptoms in advanced colorectal cancer, with 33% of MABp1-treated patients achieving clinical benefits versus 19% with placebo

Targeted Therapies

  • EGFR-targeted therapies including cetuximab and panitumumab are FDA approved for metastatic colorectal cancer
  • Other monoclonal antibody agents under investigation include matuzumab, MDX-447, nimutozumab, and mAb806
  • EGFR tyrosine kinase inhibitors being evaluated include erlotinib, gefitinib, EKB-569, lapatinib, PKI-166, and canertinib
  • Anti-VEGF antibody therapy in combination with chemotherapy improves patient survival

Metabolic Regulation

  • Oxymatrine exhibits anti-metastatic activity by inhibiting PKM2-mediated aerobic glycolysis
  • It suppresses production of ATP, pyruvate, and lactate while inhibiting glucose transporter 1 (GLUT1)
  • Oxymatrine significantly inhibits liver metastasis of CRC cells in mice

Genetic and Epigenetic Mechanisms

  • Long noncoding RNAs (lncRNAs) play roles in biological processes in CRC and may serve as prognostic and diagnostic biomarkers
  • Noncoding RNAs (ncRNAs) including microRNAs, long noncoding RNAs, and circular RNAs modulate TGF-β signaling
  • Vitamin D intake is strongly negatively associated with DKK1 methylation, suggesting vitamin D may alter CRC risk by mediating extracellular Wnt inhibition

Novel Therapeutic Approaches

  • Probiotics can enhance the antitumor effect of 5-fluorouracil (5-FU) chemotherapy
  • Thymol creates oxidative stress through ROS production, DNA damage, and mitochondrial damage
  • Thymol upregulates expression of PARP-1, p-JNK, cytochrome-C and caspase-3 proteins, triggering mitoptotic cell death

These emerging mechanisms represent promising avenues for developing more effective colorectal cancer treatments, with particular emphasis on targeting specific molecular pathways and combining therapies to enhance efficacy.

Recent Studies

Recent Colorectal Cancer Studies: Interventions, Safety and Efficacy

Taurolidine vs. Povidone-iodine Study

Napabucasin Study

Columbamine Study

Peer Support Study

Systematic Review of 242 Trials (1967-2007)

Multi-centre 2×2 Factorial Randomized Controlled Trial

Drug used in other indications

Based on the provided context, there is no information available about Muzastotug (ADG126) and Pembrolizumab being trialed for indications other than Colorectal Cancer.

The context does not contain any specific information about: - Muzastotug or ADG126 in any clinical trials - Any combination therapy involving Muzastotug (ADG126) and Pembrolizumab - Intervention models for trials involving these specific drugs - Other indications being studied for this drug combination

The only mention of Pembrolizumab in the context is related to its use as "an anti-PD-1 immune checkpoint inhibitor" in relation to colorectal cancer with mismatch repair deficiency, but no information is provided about its combination with Muzastotug or trials in other indications.

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