First, the association betweensleepdurationwith both morbidity and mortality, with a focus oncardiovasculardisease, is reviewed. Then, a brief review of the potential role of proinflammatory markers incardiovasculardiseaseis presented.
The effect of extremesleepdurationon the risk ofcardiovascularand cerebrovasculardiseases(CCDs) remains debatable.A significant U-shaped relationship was revealed of nighttimesleepdurationwith eithercardiovascularor cerebrovasculardisease.
Learn how qualitysleepsupportscardiovascularrecovery and lowersdiseaserisk.
This study aimed to identifysleepdurationtrajectories in a healthy population and examine their associations with incidentcardiovasculardisease(CVDs) in the Eastern Mediterranean Region (EMR).
One plausible mechanism by which longsleepcould contribute tocardiovasculardiseaseis via arousals fromsleep. It has been shown that longsleepershave more fragmentedsleepthan average or short-durationsleepers(25).
CVDs,cardiovasculardiseases; MR, Mendelian randomization; AIS, any ischemic stroke; ICH, primary intracranial hemorrhage. supported that shortsleepdurationrather than longsleepdurationis associated with the risk of CAD or HF.
First, the association betweensleepdurationwith both morbidity and mortality, with a focus oncardiovasculardisease, is reviewed. Then, a brief review of the potential role of proinflammatory markers incardiovasculardiseaseis presented.
As we can see from the illustration, Sleep Duration And Cardiovascular Disease has many fascinating aspects to explore.
Figure 4 Subgroup analyses for the associations of totalsleepdurationandPA trajectories with the risk ofcardiovasculardiseases.(A): Totalsleepduration; (B): PA. The moderatesleepdurationtrajectory and high stable PA trajectory were used as references, respectively.
First, the association betweensleepdurationwith both morbidity and mortality, with a focus oncardiovasculardisease, is reviewed. Then, a brief review of the potential role of proinflammatory markers incardiovasculardiseaseis presented.
