By combining the cellular turnover rates thus calculated with the mean mass of different cell types3,59, we were able to calculate the contribution of various tissue types to the whole human cellular mass turnover, presented in Fig.
We sought to establish -cell mass, -cell apoptosis, and -cell replication in humans in response to obesity and advanced age. Research design and methods. We examined human autopsy pancreas from 167 nondiabetic individuals 20102 years of age.

We mapped species richness and turnover of all rodents and the principal clades to identify the main predictors of diversity patterns. Species richness was highest in the Andes, with a secondary hotspot in Atlantic Forest and some regions of considerable richness in Amazonia.

protein turnover in rodents of different sizes. Equations and.The six-pool model of protein turnover in a rodent was able. to duplicate data (Bernier and Calvert 1987, Obled et al. 1991) of changes in specic radioactivities in free amino acid.

he incidence of type 2 diabetes in-creases with obesity and aging (1). There is a decit in b-cell mass with increased b-cell apoptosis in type 2 diabetes (2). Although there are numer-ous studies of changes in b-cell mass and turnover in rodents, inevitably the data is much more...
Maintenance of pancreatic cell mass results from a dynamic balance of neogenesis, proliferation, and apop-tosis (1). These processes are adaptive since cells can proliferate physio-logically in postnatal life (during growth or pregnancy), in response to injury...